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doi:10.1242/jeb.058214
The ability to follow transcellular pathways and induce inflammation can vary greatly among meningococcal strains and, therefore, the discrepancies found in the literature between transcellular and paracellular pathways for mechanisms for crossing the nasopharyngeal barrier may be due to the use of different strains in the various studies, as well as different cellular models
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Microbial metabolites in multiple sclerosis: Implications for pathogenesis and treatment
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